Publication:
Mitophagy and Oxidative Stress in Cancer

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dc.authorscopusid60045725900
dc.authorscopusid59124568000
dc.authorscopusid57219414682
dc.contributor.authorSezgin, F.G.
dc.contributor.authorTorunoglu, E.İ.
dc.contributor.authorAytar, E.C.
dc.date.accessioned2025-12-11T00:33:58Z
dc.date.issued2025
dc.departmentOndokuz Mayıs Üniversitesien_US
dc.department-temp[Sezgin] Fatma Gönül, Ondokuz Mayis Üniversitesi, Samsun, Turkey; [Torunoglu] Emine Incilay, Necmettin Erbakan Üniversitesi, Meram, Konya, Turkey; [Aytar] Erdi Can, Department of Horticulture, Usak University, Usak, Turkeyen_US
dc.description.abstractThis chapter highlights mitochondria's central role in eukaryotic cells for energy production via aerobic respiration, synthesizing ATP. Additionally, mitochondria contribute to lipid metabolism, calcium signaling, and ROS production. Mitophagy is crucial for maintaining homeostasis by degrading damaged mitochondria, with its four stages detailed. The link between mitochondrial damage and diseases is explored, along with the significance of mitophagy in cancer cells and potential treatments. This chapter provides insight into mitochondria's biological significance and their role in disease pathogenesis. © 2025 by IGI Global Scientific Publishing. All rights reserved.en_US
dc.identifier.doi10.4018/979-8-3693-3836-0.ch001
dc.identifier.endpage20en_US
dc.identifier.isbn9798369338360
dc.identifier.isbn9798369338377
dc.identifier.scopus2-s2.0-105013927169
dc.identifier.startpage1en_US
dc.identifier.urihttps://doi.org/10.4018/979-8-3693-3836-0.ch001
dc.identifier.urihttps://hdl.handle.net/20.500.12712/37495
dc.language.isoenen_US
dc.publisherIGI Globalen_US
dc.relation.publicationcategoryKitap Bölümü - Uluslararasıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.titleMitophagy and Oxidative Stress in Canceren_US
dc.typeBook Parten_US
dspace.entity.typePublication

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