Publication:
Pulmonary Amyloidosis

dc.authorscopusid55849360900
dc.authorscopusid6701685009
dc.authorscopusid22947045900
dc.contributor.authorCengiz, K.
dc.contributor.authorŞahan, C.
dc.contributor.authorGüner, E.
dc.date.accessioned2025-12-11T02:01:26Z
dc.date.issued2000
dc.departmentOndokuz Mayıs Üniversitesien_US
dc.department-temp[Cengiz] Kuddusi, Tip Fakultesi, Ondokuz Mayis Üniversitesi, Samsun, Turkey; [Şahan] Cem, Tip Fakultesi, Ondokuz Mayis Üniversitesi, Samsun, Turkey; [Güner] Ertuǧrul, Tip Fakultesi, Ondokuz Mayis Üniversitesi, Samsun, Turkeyen_US
dc.description.abstractAmyloidosis results from the deposition of insoluble, fibrous amyloid proteins, nearly always in the extracelluler species of organs and tissues. Amyloid deposition may occur in association with inflammatory, hereditary or neoplastic conditions. It may develop as part of a disorder of immunoglobulins. Virchow first described amyloid involving the lungs in 1857. Pulmonary amyloidosis may be localized to be respiratory tract or may be part of a widespread process involving many organs. Localized pulmonary amyloidosis is a rare disorder that appears in four forms; tracheobronchial, nodular parenchymal, diffuse interstitial and senile pulmonary amyloidosis.en_US
dc.identifier.endpage203en_US
dc.identifier.issn1300-2996
dc.identifier.issue3en_US
dc.identifier.scopus2-s2.0-0034496637
dc.identifier.scopusqualityN/A
dc.identifier.startpage198en_US
dc.identifier.urihttps://hdl.handle.net/20.500.12712/47599
dc.identifier.volume17en_US
dc.identifier.wosqualityN/A
dc.language.isotren_US
dc.relation.ispartofOndokuz Mayis Universitesi Tip Dergisien_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectAmyloidosisen_US
dc.subjectPulmonary Involvementen_US
dc.titlePulmonary Amyloidosisen_US
dc.title.alternativePulmoner Amiloidozen_US
dc.typeArticleen_US
dspace.entity.typePublication

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